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Which spinal artery is more prone to infarction?

Which spinal artery is more prone to infarction?

Injury to an extravertebral feeder artery or the aorta (eg, due to atherosclerosis, dissection, or clamping during surgery) causes infarction more commonly than do intrinsic disorders of spinal arteries. Thrombosis is an uncommon cause, and polyarteritis nodosa is a rare cause.

What is spinal cord infarction?

Spinal cord infarction is a stroke either within the spinal cord or the arteries that supply it. It is caused by arteriosclerosis or a thickening or closing of the major arteries to the spinal cord.

Can a human spinal cord heal itself?

Unlike tissue in the peripheral nervous system, that in the central nervous system (the spinal cord and brain) does not repair itself effectively.

Are there any studies on spinal cord infarction?

Joshua Z. Willey, J.P. Mohr, in Stroke (Fifth Edition), 2011 Spinal cord infarction, although rare, remains incompletely studied outside the cardiothoracic and vascular surgery literature. Its incidence is unknown, and no large epidemiologic study has been conducted.

When to treat biphasic spinal cord infarction?

Treatment of these risk factors is essential in the acute phase. Biphasic spinal cord strokes are seen in one-fifth of the patients. These present with acute or transient sensory spinal cord deficits often preceded by radiating pain between the shoulders, and should be considered and treated as imminent spinal cord strokes.

Can a spinal cord infarction be unilateral?

In some instances, the adjacent vertebral body demonstrates infarction, although this is not a common finding 5,7 . Usually, involvement of the posterior spinal arteries is unilateral (as posterior spinal arteries are paired) and are usually confined to the dorsal columns 5 .

What are the effects of posterior spinal artery infarction?

Posterior spinal artery territory infarction causes selective loss of proprioception or of all sensory modalities (due to dorsal horn involvement) with relative preservation of motor function. 10 Deficits are often asymmetric. Posterior spinal artery ischemia is uncommon because of the extensive collateral network in the dorsal cord.

Who are more likely to have spinal infarction?

Spinal cord infarction patients are younger and more often women compared to cerebral infarction patients. Traditional cerebrovascular risk factors are less relevant in spinal cord infarction.

Treatment of these risk factors is essential in the acute phase. Biphasic spinal cord strokes are seen in one-fifth of the patients. These present with acute or transient sensory spinal cord deficits often preceded by radiating pain between the shoulders, and should be considered and treated as imminent spinal cord strokes.

Can a spinal cord infarction cause a central cord syndrome?

Position and vibration sensation, conducted by the posterior columns, and often light touch are relatively spared. If the infarct is small and affects primarily tissue farthest away from an occluded artery (toward the center of the cord), a central cord syndrome is also possible. Neurologic deficits may partially resolve after the first few days.

How is the diagnosis of spinal cord infarction made?

Diagnosis. Infarction is suspected when severe back pain and characteristic deficits develop suddenly. Diagnosis of spinal cord infarction is by MRI. Acute transverse myelitis, spinal cord compression, and demyelinating disorders may cause similar findings but are usually more gradual and are excluded by MRI and by CSF analysis.